The many substrates and functions of ATM

As its name suggests, the ATM - 'ataxia-telangiectasia, mutated' - gene is responsible for the rare disorder ataxia-telangiectasia. Patients show various abnormalities, mainly in their responses to DNA damage, but also in other cellular processes. Although it is hard to understand how a single gene product is involved in so many physiological processes, a clear picture is starting to emerge.
Publisher
NATURE PUBLISHING GROUP
Issue Date
2000-12
Language
ENG
Keywords

ATAXIA-TELANGIECTASIA GENE; NIJMEGEN BREAKAGE SYNDROME; DEPENDENT PROTEIN-KINASE; DNA-DAMAGE RESPONSE; P53-DEPENDENT G(1) ARREST; CELL-CYCLE CHECKPOINTS; IONIZING-RADIATION; SACCHAROMYCES-CEREVISIAE; INDUCED PHOSPHORYLATION; MEIOTIC RECOMBINATION

Citation

NATURE REVIEWS MOLECULAR CELL BIOLOGY, v.1, no.3, pp.179 - 186

ISSN
1471-0072
DOI
10.1038/35043058
URI
http://hdl.handle.net/10203/74824
Appears in Collection
BS-Journal Papers(저널논문)
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