EXPANSION OF IMMUNOSENESCENT T CELL FRACTION IN CHRONIC KIDNEY DISEASE PATIENTS: EXPLORING NEW PATHOGENIC MECHANISMS OF INCREASED ARTERIAL STIFFNESS

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dc.contributor.authorYoun, Jong-Chanko
dc.contributor.authorYu, Hee Taeko
dc.contributor.authorKim, Hyeon Changko
dc.contributor.authorChoi, Suk-Wonko
dc.contributor.authorHan, Seong-Wooko
dc.contributor.authorRyu, Kyu-Hungko
dc.contributor.authorShin, Eui-Cheolko
dc.contributor.authorPark, Sunghako
dc.date.accessioned2017-07-04T02:26:27Z-
dc.date.available2017-07-04T02:26:27Z-
dc.date.created2017-06-23-
dc.date.created2017-06-23-
dc.date.issued2016-09-
dc.identifier.citationJOURNAL OF HYPERTENSION, SUPPLEMENT, v.34-
dc.identifier.issn0952-1178-
dc.identifier.urihttp://hdl.handle.net/10203/224578-
dc.description.abstractObjective: Chronic kidney disease (CKD) is associated with increased arterial stiffness, which is a well-known predictor of future cardiovascular events. However, the underlying mechanism of arterial stiffening in CKD is not well known. Accelerated immune aging, characterized by expansion of immunosenescent T cell fraction might be involved in the pathogenesis of arterial stiffening in CKD. We aimed to evaluate the relationship between arterial stiffness and immunosenescent T cell (CD8+CD57+ or CD8+CD28- T cell) fraction in patients with CKD-
dc.languageEnglish-
dc.publisherLippincott Williams & Wilkins Ltd.-
dc.titleEXPANSION OF IMMUNOSENESCENT T CELL FRACTION IN CHRONIC KIDNEY DISEASE PATIENTS: EXPLORING NEW PATHOGENIC MECHANISMS OF INCREASED ARTERIAL STIFFNESS-
dc.typeArticle-
dc.type.rimsART-
dc.citation.volume34-
dc.citation.publicationnameJOURNAL OF HYPERTENSION, SUPPLEMENT-
dc.identifier.doi10.1097/01.hjh.0000500985.37648.66-
dc.contributor.localauthorShin, Eui-Cheol-
dc.contributor.nonIdAuthorYoun, Jong-Chan-
dc.contributor.nonIdAuthorYu, Hee Tae-
dc.contributor.nonIdAuthorKim, Hyeon Chang-
dc.contributor.nonIdAuthorChoi, Suk-Won-
dc.contributor.nonIdAuthorHan, Seong-Woo-
dc.contributor.nonIdAuthorRyu, Kyu-Hung-
dc.contributor.nonIdAuthorPark, Sungha-
dc.description.isOpenAccessN-
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MSE-Journal Papers(저널논문)
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